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General information about this database and other resources for identifying and treating goat diseases
Diseases from all over the world are included
USAID and other agencies
are providing computers to extension agents in small villages all over
the world, enabling them to access agricultural materials. At last
count people from 93 countries were using this site. In addition,
people are traveling and working all over the world, and disease is
spreading from country to country.
Reader Responsibility
This database was created by a person
who is not a veterinarian. There is no warranty that the
materials are correct. Every one who uses this database should
read all of the resources under "Summarized From..." and should consult
the other links provided in the right column to verify the accuracy of
the material before using it.
Veterinary Texts
Smith and Sherman's Goat Medicine, Lippincott,
Wilkins and Williams, 1994 is an excellent veterinary text and is highly recommended
for any person providing health care to goats. (Note:
this book has been updated since the writing of this section, so all
page numbers cited here apply to the 1994, not the 2009 edition.)
Goat anatomy illustrations
Veterinary databases or articles created by licensed veterinarians
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Item
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Document Page #
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Site Screen
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Goat Formulary
(drugs and drug dosages for goats
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44-53
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50-53
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Goat Differential Diagnosis List
(Possible diagnoses by symptoms)
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54-91
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60-97
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Goat Laboratory Result Differentials
(Test results and what the cause might be)
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144-152
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150-158
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Goat Toxins
(Body system involved and potential causes)
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153-156
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159-162
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Goat Nutrient Imbalances
(body system involved and possible causes)
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157-158
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163-164
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Caprine Theriogenology
(a vet specialty in reproduction)
(Reproduction fact sheet)
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198-199
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204-205
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Caprine Reproduction
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200-207
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206-213
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Dairy Goat Biosecurity
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208-209
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214-215
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Parasite Control .
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227-237
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233-244
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Ultrasounding Goats .
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238- 243
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244-249
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Nutrition
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244-262
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250-269
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Glossary of Diseases
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270-288
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276-294
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Abbreviation list
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289-290
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295-29
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Langston University's 2010 Field Day Notes: Meat Goat Herd Health and Prevention (Dr. Lionel Dawson) are an excellent resource for basic goat care. The article includes tables of vaccinations, anthelmintics (dewormers)
and common drugs with the currently recommended goat dosages, milk and meat
withhold times. They are written for meat goats,
but most of the material applies to dairy goats as well. http://www.luresext.edu/goats/library/field/dawson10.pdf
The following sites
contain useful medical information, but are not written by veterinarians:
Milk Producers: READ THIS NOTICE
Many drugs are not approved for use in goats, so the label directions
for dosage, route of administration and meat and milk withhold times,
meant for other livestock, may be incorrect and dangerous for
your goats. In addition, if you use the wrong meat or milk
withhold time, you can end up with drugs in the food chain, and this
can result in huge financial losses as well as the potential loss of
your dairy license. By law, all drugs which are not approved for goats, must
be given under the supervision of a veterinarian who works with your herd
on a regular basis.
Do not treat your goats without consulting with your local veterinarian
first. For more information see the section on "Extra-label drug
use" on the Medical pages, and read the "Veterinary Issues" and
"Insurance" sections of the Setting Up Your Dairy page.
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| The Disease Database A-B |
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Acariasis (see Mange)
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Akabane
(Summarized from Smith and Sherman, Goat Medicine, at p.
82-85, and Haskell, Small Ruminant Clinical Diagnosis and Therapy, at p. 270.
)
Causative Agent: Virus passed by biting midge and some
types of mosquitoes. Found in Asia, Africa, Middle East and Australia.
Clinical Signs: Disease is inactive in non-pregnant
does. A pregnant doe that is infected, may be normal, but if a doe is
infected between days 30 and 50 of their pregnancy, the kids are often
malformed. If the doe is infected in the last 100 days of pregnancy,
kids are normal.
Stillborn fetuses, or newborns that die shortly after
birth, may have birth defects such as arthrogryposis (rigid fixation of the
joints; usually in flexion but occasionally in extension) and hydrancepathy
(kids have neurological deficits such as being dull, blind and having weak
muscles).
Treatment: None.
Prevention: Vaccination in done in areas that have had
previous infections.
Vaccinate before breeding season.
Contagious To Humans: no
Contagious to Other Goats: no
Diagnosis: clinical presentation and virus
isolation from placenta.
Links:
Akabane Fact Sheet
http://www.cfsph.iastate.edu/Factsheets/pdfs/akabane.pdf
Akabane Power Point Slide Show
http://www.cfsph.iastate.edu/Factsheets/pdfs/akabane.pdf
Akabane Speaker's notes
http://www.cfsph.iastate.edu/DiseaseInfo/notes/akabane.pdf
Akabane Vaccine
http://apps.cfsph.iastate.edu/Vaccines/disease_list.php?diseaseID=4 |
Alopecic
Exfoliative Dermatitis
(Summarized from Smith and Sherman, Goat Medicine p. 38)
Causative Agent: Psoriasiform dermatitis (psoriasis). May be hereditary.
Clinical Signs: Hair loss, scales and crusts around
the eyes, lips, chin, ears, belly and pubic area of Pygmy goats.
Treatment: Steroids
Prevention: none
Contagious To Humans: no
Contagious to Other Goats: no
Links: |
Anthrax
(Summarized from Smith and Sherman, Goat Medicine p.208-209 and Haskell, Small Ruminant Clinical
diagnosis and Therapy p. 270)
Causative Agent: Bacillus anthracis. Ability to
produce spores that can live 50 years. Endemic in many tropical and
subtropical areas of the world.
Rare to find in goats in the U.S. Cases have been
found in Texas. Also, a S. Carolina worker got it from handling
cashmere goat hair imported from western Asia.
Normal mode of transmission: Ingestion of spores while
grazing
Clinical Signs: Salivating, extremely depressed
attitude, head hanging down. Over a period of 1-2 days become unable to
stand, and die slowly. See a failure of the blood to clot. Will see
bloody discharge from the nose and mouth after death. Do not touch or
open carcass. DO NOT DO POST MORTEM EXAM. Veterinarian must
report disease to state and federal authorities and they will examine and
remove carcass.
Treatment: Antiserum and /or antibiotics including
tetracyclines, streptomycin and penicillin may be tried in early
stages.
Prevention: Vaccines are available and are used in areas
of the world where this disease is common. They are given once a year.
Contagious To Humans: yes
Contagious to Other Goats: yes
Links:
Smith and Sherman, Goat
Medicine p.208-209
Haskell, Small Ruminant Clinical diagnosis and Therapy p.
270
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BDV (Border Disease, Hairy shaker disease)
(Summarized from Smith and Sherman, Goat Medicine,
p.139-140 (basic information), 187 (retinal changes), 427 and 139 (abortion). Also Haskell, Small Ruminant
Clinical diagnosis and Therapy p. 270)
Causative Agent: Border disease virus (BDV), Found in
sheep and goats. Related to Bovine Virus Diarrhea (BVD) virus .
Clinical Signs: Adult does show no clinical signs except early abortion,
fetal resorption, mummification, infertility and stillbirths. The disease is spread by aerosol inhalation
or ingestion.
Kids are infected through the placenta of an infected
mother. If kids live, they will be
weak, have a pattern of rhythmic tremors or shaking starting in the rear
legs, then the trunk and neck.
Difficulty rising, and awkward gait.
Appear weak, listless, depressed.
The Long bones of the legs may feel thinner than usual, and the head
appears to be narrow and convex in the frontal area. The disease is found in Norway only,
although goats in other countries may have antibodies against the
disease.
Note: Vets familiar with Border Disease in lambs may
expect to see changes in hair. Goat kids will not have the changes in
hair that lambs have when exposed in utero. (P. 37 Smith.)
Treatment: None.
Prevention: Isolate all does that abort regardless of
cause. Border disease virus is found
in vaginal discharges after abortion or birthing. Diagnose on history of reproductive
failure, occurrence of newborn shaker kids, and positive laboratory tests for
the virus. Maintain goats away from
cattle and sheep. Cull goats producing
shaker kids. Bovine vaccines may not
be effective.
Contagious To Humans: no
Contagious to Other Goats: -
Links:
Smith and Sherman, Goat Medicine, p. 139-140, 187 retinal
changes, 427 and 139 abortion,
Haskell, Small Ruminant Clinical diagnosis and Therapy p.
270)
Border Disease in Goats http://www.ivis.org/advances/Disease_Tempesta/Loken/ivis.pdf
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Bloat
Two types:
Frothy: caused by diets that promote froth.
Free gas bloat: caused by diets that promote excessive gas
production or by a failure to belch.
(Summarized from Smith and Sherman, Goat Medicine,
p.340 and Haskell, Small Ruminant Clinical diagnosis and Therapy p. 270.
Additional information from Dr. Emma Ewing, DVM)

Picture from John Elfick, University of Queensland.
UNESCO Goat Project for the Solomon Islands.
http://www.uq.edu.au/_School_Science_Lessons/57Goat7.GIF
Causative Agent: Accumulation of either froth or free gas
in the rumen, which causes rumen distention
Frothy bloat is usually associated with legume forages or
hay, also grazing on lush cereal grain pastures, wet grass pastures, or
high-grain diets too finely ground. The
feeding of garden greens to animals on a dry hay diet may cause bloat. Frothy
bloat can occur within hours of exposure to the offending feed or feeding
situations.
Free gas bloat occurs with grain diets, especially if the animals are not
adapted to the diet. It can also be caused by a physical obstruction in the
esophagus or other conditions such as internal abscesses, hypo-calcaemia,
pain, or peritonitis.
Clinical Signs: Goat will be anxious and uncomfortable and will stop eating.
There is a progressive distension of the abdomen. As the abdomen enlarges,
the goat becomes more uncomfortable, stamping feet, vocalizing, salivating,
urinating frequently, and moving with a stilted gait. Breathing becomes
labored, and if left untreated, animals will die.
Treatment: Act right away. Bloat can kill very
quickly.
Free gas bloat can be relieved by passing a stomach tube.
Frothy bloat: you must break the froth down first and then
pass the stomach tube.
Break down foam by giving cooking oil or mineral oil (100
cc. adult and 8 cc. to a young kid), or Bloat Guard®. Pass the feeding tube or use a calf bag and
put the tip down the esophagus. For
the calf feeding bag: If you are in the right place, you will not be able to
see the tube on the outside of the throat.
If the calf feeder bag tube is in the wrong place (may be in the
trachea or windpipe by mistake), you will hear air movement and you can see
the tube bulge out on the outside of the neck. If you are in the wrong place, remove it
and try again. With the regular feeding tube, you should hear gurgling sounds
if you are in the stomach, and breathing (air) movements if you are in the
lung. If you are in the lung, remove
the tube and reinsert it. make sure
you are in the stomach before releasing the liquid. If liquid is released into the lung, it may
cause pneumonia or drowning.. (See tube feeding instructions in the Medical
section) Whatever tube you use, when the tube enters the stomach area, you
should hear a big release of gas.
If the goat is not better in twenty minutes, put the oil
directly into the rumen by introducing an 18 gauge needle into the left
paralumbar fossa. (see diagram on the left.) Place your
fingertips on the goat's left side (Note: if you are facing him, it is your
right side), halfway between the ribs and hips, and about 1/4 of the way down
from the top line. This soft, hollow area high on either side below the loin
is the paralumbar fossa. You
should feel a hard mass. This is the rumen contents. (Kinne ] Note:
this procedure may cause peritonitis and rumen dysfunction so try everything
else first. If you must do it, then
give 3-5 days of broad spectrum antibiotics afterward.
Alternative treatment from Emma Ewing, DVM:
(Kid) 2T baking soda in the same amount of warm water they
are getting in their bottle, and a dash of dish soap.
(Adult) 1/4 c. backing soda, 2 cups water and a squirt of
dish soap.
If no relief in 20 minutes. then puncture as directed
above with 18 gauge needles and inject 8cc, of cooking oil or mineral oil for
a kid, or 100 cc. for an adult..
Prevention:
1) Control intake of legume pasture and cereal crops in
spring, by feeding hay first, then putting on pasture, and gradually
increasing time on pasture.
2) Make sure goats can’t get into feed bin.
3) Feed all grains whole, halved or quartered. No finer than that.
Contagious To Humans: No
Contagious to Other Goats: No
Links:
Smith and Sherman, Goat Medicine,
p.340.
Haskell, Small Ruminant Clinical diagnosis and Therapy p.
270.
Bloat http://www.sheepandgoat.com/news/august2004.html#IPM
Bloat: Rumen Motility and Bloat Diagnosis http://kinne.net/rum-mot.htm |
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Blue Tongue
(Summarized from Smith and Sherman Goat Medicine, at
p. 294-296 andHaskell, Small Ruminant Clinical Diagnosis and Therapy at p.
270)

Erosion and reddening of the mucosa (sheep photo)
Photos taken by the Plum Island Animal Disease Center, and
contributed to the CFSPH website at: http://www.cfsph.iastate.edu/DiseaseInfo/ImageDB/imagesBT.htm
Used with permission of CFSPH.
Light coronary band, small bruises (petechia) in hoof
(sheep photo)
Photos taken by the AFIP and contributed to the CFSPH
website:http://www.cfsph.iastate.edu/DiseaseInfo/ImageDB/imagesBT.htm
Used with permission of CFSPH.
Causative Agent: Orbi-virus carried by biting midges,
sand flies, no see-ums, gnats, sheep keds, ticks and biting flies. The
infection is usually sub-clinical in goats. That is, they usually have
it in their body, but don't get a full blown case. There are
exceptions, and there have been epidemics reported where goats get full-blown
disease. Disease comes in warm months when insects are present. the
virus may cross the placenta to fetuses, and it can be found in semen. .
Clinical Signs: In the goat, most cases are limited to mild depression,
temporary loss of appetite, fever up to 105 degrees (40.5 C), and engorgement
of blood on the tissues of the mouth and nose.
In more severe cases, fever and loss of appetite for 3-4
days, followed by engorgement of the lining of the mouth, with loss of tissue
on the tongue, lips and gums, that can become open sores, and excess
salivation. The face swells and there is a nasal discharge.
Sometimes the goat will get diarrhea.
Inflammation. swelling and blood engorgement develops in
the coronal band above the hoof, leading to lameness. Walnut-sized
eruptions may appear on the skin all over the body. This presentation
is found only in goats. The disease runs it's course in 8-12 days.
Treatment: There is no treatment to cure, but
antibiotics may be given to prevent secondary infection.. Feed
separately so they don't have to compete for food. Avoid sunlight.
Quarantine the animal. Report to State Veterinarian.
Prevention: Quarantine all new stock brought onto the
farm. During warm months, animals should not graze in low-lying areas
which may be wet, and may harbor the insects that carry the disease.
Contagious To Humans: no
Contagious to Other Goats: no
Diagnose by: Paired sera titers, virus isolation,
post-mortem exam
Differentiate from: Foot and Mouth disease, goat pos, Orf,
photosensitivity, Akabane.
Links:
Smith and Sherman, Goat Medicine, at p. 294-296
Haskell, Small ruminant Clinical Diagnosis and Therapy at
p. 270
Bluetongue Fact Sheet
http://www.cfsph.iastate.edu/Factsheets/pdfs/bluetongue.pdf
Bluetongue Power Point Slide Show
http://www.cfsph.iastate.edu/DiseaseInfo/ppt/bluetongue.ppt
Bluetongue Speaker's notes
http://apps.cfsph.iastate.edu/Vaccines/disease_list.php?diseaseID=8
Bluetongue Vaccine
http://apps.cfsph.iastate.edu/Vaccines/disease_list.php?diseaseID=8
Bluetongue http://www.oie.int/eng/maladies/fiches/a_A090.htm
Bluetongue
http://www.fao.org/ag/againfo/subjects/en/health/diseases-cards/bluetongue.html |
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Brucellosis
(Summarized from Smith and Sherman Goat Medicine, at p.
423-424 (basic information), 444 (orchitis in bucks), and Haskell, Small Ruminant Clinical
Diagnosis and Therapy at p. 271)
Causative Agent: Brucella melintensis and Brucella abortis
gram negative coccobacillus. Organism is excreted in milk, urine,
feces, fetuses, and can be found in placenta and vaginal discharges for up to
2-3 months after birthing. Enters body through nose or through direct
penetration through skin. Kids born alive to infected does are infected
and capable of spreading the organism.
Clinical Signs: Mid to late gestation abortions in
stressed herds, often with nutritional parasitic disease left uncontrolled,
though a mid to late abortion storm can be expected even in a well managed
herd..
Treatment: Test and slaughter positive goats. Report to
State Veterinarian.
Prevention: Vaccinate goats, especially kids 3-8
months of age in areas where disease is commonly found: Middle East, India,
China, parts of Latin America..
Contagious To Humans: Yes.
Contagious to Other Goats: Yes.
Diagnosis: confirmed by culture from placenta, fetus or
vaginal discharge
Links:
Smith and Sherman Goat Medicine, at p. 423-424, 444
(orchitis in bucks).
Haskell, Small Ruminant Clinical Diagnosis and Therapy at
p. 271
Brucellosis Fact Sheet
http://www.cfsph.iastate.edu/Factsheets/pdfs/brucellosis.pdf
Brucellosis Speaker's Notes
http://www.cfsph.iastate.edu/DiseaseInfo/notes/Brucellosis.pdf
Brucellosis Power Point Slide Show
http://www.cfsph.iastate.edu/DiseaseInfo/ppt/brucellosis.ppt
Brucellosis Vaccine
http://apps.cfsph.iastate.edu/Vaccines/disease_list.php?diseaseID=13
Brucellosis
http://www.fao.org/ag/againfo/subjects/en/health/diseases-cards/brucellosi-ov.html
Haskell, Small Ruminant Clinical Diagnosis and Therapy, at
p. 271, http://www.rmncsba.org/smallruminant.pdf
Smith and Sherman, Goat Medicine, at p. 423-424, 444.
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